After Gleeson read more.

After Gleeson, the , as almost, as almost none of the mice survived in this strain until adulthood that the deaths may be on epilepsy recorded. Scientists electroencephalograms, which measure electrical activity produced by the firing of neurons in the brain, and found severe epilepsy in all mice tested Even more surprising was the site the epileptic focus read more more info . Or website from which the seizures were generated – that was beneath the surface of the brain, the hippocampus is.

Cortical dysplasia, ie disordered development of the cerebral cortex, in in 25 to 40 % of children with the most severe and difficult to treat forms of epilepsy. These children have often come to the attention of specialists due to stagnation in the acquisition of language and balance skills and accompanying epilepsy. How small muscle spasms or eyelid fluttering – to dramatic whole body, tonic-clonic spasms , until essential function of the body, the symptoms of these children appear ranging from very subtle. – The Gleeson team of researchers Geraldine Kerjan, PhD and Hiroyuki Koizumi studied, since a condition called ‘lissencephaly. ‘ Children with lissencephaly have a smooth brain surface. The normal hills and valleys, the missing characteristic of the human brain The researchers have been successful recently in the development of a mouse model that some of the features of this disease, was usually the first step in understanding the cause of a genetic disorder. But the severe epilepsy with with lissencephaly in in any of the preceding animals, so that the team met kept removing gene after gene, up up on a tribe of epilepsy. – Doublecortin: ‘We studying the gene ‘, ‘which is defective in some forms of epilepsy and mental retardation in humans,’said Kerjan, lead author of the study. ‘However, only epilepsy epilepsy discover a combination of two of the genes in the doublecortin family away us. ‘.

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